monoclonal rabbit anti gr antibody (Cell Signaling Technology Inc)
Structured Review

Monoclonal Rabbit Anti Gr Antibody, supplied by Cell Signaling Technology Inc, used in various techniques. Bioz Stars score: 86/100, based on 1 PubMed citations. ZERO BIAS - scores, article reviews, protocol conditions and more
https://www.bioz.com/product/monoclonal+anti+gr/pmc13064270-124-57-61
Average 86 stars, based on 1 article reviews
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1) Product Images from "Alpha-1 antitrypsin–glucocorticoid receptor axis: a new pathway in immune modulation"
Article Title: Alpha-1 antitrypsin–glucocorticoid receptor axis: a new pathway in immune modulation
Journal: Molecular Medicine
doi: 10.1186/s10020-026-01470-z
Figure Legend Snippet: Endogenous AAT and GR co-localize in human PBMCs. Co-staining of adherent human blood PBMCs with anti-AAT ( red ) and polyclonal ( A ) or monoclonal ( B ) anti-GR ( green ) antibodies reveals co-localization of AAT and GR in the cytoplasm. Nuclei were stained with DAPI. Images were taken using a confocal laser scanning microscope (Olympus FluorView 1000) equipped with a 60× oil-immersion objective. Scale bars represent 20 μm
Techniques Used: Staining, Laser-Scanning Microscopy
Related Articles
Incubation:Article Title: Glucocorticoids stimulate hypothalamic dynorphin expression accounting for stress-induced impairment of GnRH secretion during preovulatory period. Article Snippet: Stress-induced reproductive dysfunction is frequently associated with increased glucocorticoid (GC) levels responsible for suppressed GnRH/LH secretion and impaired ovulation.. Besides the major role of the hypothalamic kisspeptin system, other key regulators may be involved in such regulatory mechanisms.. Herein, we identify dynorphin as a novel transcriptional target of GC. Article Title: Glucocorticoid receptor signaling in astrocytes is required for aversive memory formation Article Snippet: Serial 40-μm-thick coronal sections from CTRL and GR astroKO mice obtained with vibratome were processed in parallel. .. Free-floating sections were rinsed in PBS, incubated with 10% normal goat serum in PBS with 0.2% Triton X−100 (PBS-Tx) for 90 min at room temperature (RT) and incubated overnight at RT with antibodies: polyclonal anti-S100β (1:250, 287004, Synaptic Systems) or a monoclonal anti-NeuN (1:200, MAB377, Merck Millipore) and a Article Title: Acute stress responsiveness of the neurotrophin BDNF in the rat hippocampus is modulated by chronic treatment with the antidepressant duloxetine. Article Snippet: Equal amounts of protein were run under reducing conditions on SDS–polyacrylamide gels (14% SDS–PAGE for BDNF and 8% SDS–PAGE for glucocorticoid receptor (GR), a-calcium/calmodulin-dependent protein kinase II (aCaMKII), CREB, methyl CpG-binding protein 2 (MeCP2), mitogen-activated protein kinase/extracellular signal-regulated kinase kinase (MEK)) and then electrophoretically transferred onto PVDF (for BDNF) or nitrocellulose (for the other proteins) membranes (Bio-Rad). .. The blots were blocked with 10% nonfat dry milk and then incubated with the following primary antibodies: anti-BDNF polyclonal antibody (1 : 1000, 41C, overnight; Santa Cruz Biotechnology, Santa Cruz, CA, USA) able to recognize both the mature form of the neurotrophin (mBDNF; 14 kDa) and its precursor (proBDNF; 32 kDa); Blocking Assay:Article Title: Glucocorticoids stimulate hypothalamic dynorphin expression accounting for stress-induced impairment of GnRH secretion during preovulatory period. Article Snippet: Stress-induced reproductive dysfunction is frequently associated with increased glucocorticoid (GC) levels responsible for suppressed GnRH/LH secretion and impaired ovulation.. Besides the major role of the hypothalamic kisspeptin system, other key regulators may be involved in such regulatory mechanisms.. Herein, we identify dynorphin as a novel transcriptional target of GC. Saline:Article Title: Glucocorticoids stimulate hypothalamic dynorphin expression accounting for stress-induced impairment of GnRH secretion during preovulatory period. Article Snippet: Stress-induced reproductive dysfunction is frequently associated with increased glucocorticoid (GC) levels responsible for suppressed GnRH/LH secretion and impaired ovulation.. Besides the major role of the hypothalamic kisspeptin system, other key regulators may be involved in such regulatory mechanisms.. Herein, we identify dynorphin as a novel transcriptional target of GC. |